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Grand Junction, Colorado

Copyright 2010 - 2017 The Food Alternative
All rights reserved
Renee’ Calder, CNT, MNT
(970) 201-5723

Applying the Science of Nutrition to the
Art of Healing on the Western Slope of Colorado
Grand Junction, Colorado

Copyright 2010 - 2018 The Food Alternative
All rights reserved
Renee’ Calder, CNT, MNT
(970) 201-5723
TheFoodAlternative.com

Applying the Science of Nutrition to the
Art of Healing on the Western Slope of Colorado
Disclaimer:  Nutrition Therapy is not intended as a diagnosis, a treatment, a prescription, or a cure for any disease either mental or physical,
and is not intended as a substitute for regular medical care. Nutrition therapy does provide nutritional evaluation, balanced diet planning,
nutritional supplement suggestions and lifestyle recommendations for the purpose of enhancing health.
Alzheimer's Diesease is a common type of dementia, or decline in
intellectual function. It affects more than 4.5 million people in the
United States. It is characterized by progressive mental
deterioration to such a degree that it interferes with one’s ability
to function socially and at work. Memory and abstract thought
processes are impaired.         
      Alzheimer’s disease is considered an irreversible, progressive
disorder by the medical establishment. Deterioration in critical
areas of the brain may precede symptoms by as much as twenty to
forty years. A decline in the sense of smell often occurs as much as
two years prior to diagnosis. As the disease progresses, there is
severe memory loss, particularly in short-term memory. The person
may recall past events but be unable to remember a just viewed
television show. At this stage, disorientation usually begins as well.
Dysphasia (the inability to find the right word) may occur, and
mood swings can be unpredictable and sudden. In the final stage,
Alzheimer’s creates severe confusion and disorientation, and
possibly hallucinations or delusions. Some people become violent and
angry, while others may be docile and passive. It is also in this stage
that the patient may wander without purpose, experience
incontinence, and neglect personal hygiene. Since the behavioral
symptoms of this disease result from changes in the brain, the
person neither intends to, nor can control this behavior.
      Alzheimer’s disease is a degenerative disorder that is
characterized by a specific set of physiological changes in the brain.
Nerve fibers surrounding the hippocampus, the brain’s memory
center, become tangled (neurofibrillary tangles), and information is
no longer carried properly to or from the brain. New memories
cannot be formed, and memories from earlier cannot be retrieved.
Characteristic plaques accumulate in the brain as well. These
plaques are composed largely of a protein-containing substance
called beta-amyloid. Scientists believe that the plaques accumulate
in the damaged nerve cells.
      Homocysteine, an amino acid that forms as the result of the
breakdown of another amino acid, methionine, is a biomarker for
the development of dementia and Alzheimer’s disease. Elevated
homocysteine levels are an indication of a severe disruption of
methylation in the brains of people with Alzheimer’s disease.
Methylation deficiencies can result in severe damage to brain cells.
Which may lead to the neurological damage that occurs as the
disease advances.
      Nutrients may have a beneficial effect, and one study has
shown that vitamin E slowed the progress of some components of
the disease by up to seven months. Research also indicates that the
herb Ginko Biloba may be helpful in treating some symptoms,
although it should be used with caution as its use can lead to
excessive bleeding especially if used in conjunction with aspirin.
EFAs are essential fatty acids which are necessary for proper
nervous system function.
      Studies suggest that people with low levels of niacin (vitamin
B3) were 70 percent more likely to get Alzheimer’s than those with
higher levels in their diet. Niacin is required for cell respiration; for
energy release; for the metabolism of carbohydrates, fats, and
proteins; for the secretion of bile; for proper functioning of the
nervous system; for the synthesis of sex hormones; and for many
other processes which require this vitamin.
      No one should accept a diagnosis of Alzheimer’s disease
without first undergoing a trial of intensive nutritional therapy,
using zinc, vitamin B3, and vitamin B12 injections. Vitamin B12
functions in numerous metabolic processes affecting nerve tissue,
including the synthesis of neurotransmitters and the formation of
the insulating sheath surrounding many nerves, and it may play a
role in the fight against Alzheimer’s disease.
      Using your brain, remaining busy, writing, reading, and
learning new things are important overall factors in staying sharp
and preventing mental disorders. It may also slow down the
progression of the disease. This means keeping active and
intellectually involved, as well as getting plenty of exercise. Eat a
well-balanced diet of natural foods with plenty of fiber using oat
and rice bran. Have allergy testing performed to rule out the
possibility of environmental and/or food allergies. Avoid alcohol,
cigarette smoke, processed foods, and environmental toxins,
especially metals such as aluminum and mercury. All metals in
excess are toxic to the body. Drink 8 glasses of water per day but
do not drink tap water which may contain aluminum.
      Women with Alzheimer’s disease have been found to have
lower estrogen levels than their healthy counterparts. People who
have experienced prolonged psychological distress such as depression
and anxiety are twice more prone to this disease as well.
      Caregivers should seek counseling and support from the various
agencies and groups that are trained to help.  Support groups can
be found online, here are some links just below.
Alzheimer's Disease
ALZHEIMER'S DISEASE SUPPLEMENT LIST
Zinc 80 mg daily, in divided doses. Do not exceed 100 mg daily from
all supplements.
Acetyl-L-carnitine 500 mg twice daily
Boron 3 mg daily. Do not exceed this amount.
Coenzyme A see label.
Coenzyme Q10 100 - 200 mg daily
Folic acid see label
Lecithin granules 1 tbsp 3 times daily, before meals. Or capsules
1200 mg 3 times daily, before meals.
Multivitamin/mineral with 99 mg potassium daily.
Phosphatidyl serine 300 mg 3 times daily
SAMe 400 mg twice daily
Selenium 200 mcg daily
Trimethylglycine (TMG) 500 - 1000 mg daily, in the morning.
Vitamin A 15000 IU daily, w/ Carotenoids plus beta-carotene 25000
IU daily, plus
vitamin E 200 IU daily
Vitamin B complex injections 2 cc 3 times daily. Plus extra B6, ½ cc
weekly and B12 1cc three times weekly. If injections are not
available use vitamin B complex that includes 100 mg of each major B
vitamin. Plus extra B5 (Panthothenic acid) 100 mg three times daily.
Apple pectin see label
Calcium 1600 mg daily, at bedtime
Magnesium 800 mg daily
Amino acid complex (free form) 1000 - 2500 mg daily, 1 hr before
meals with 8 oz water
Take B6 (50 mg) and vitamin C (100 mg) to aid assimilation.
Huperzine A 100 mcg daily.
Kelp 1000 - 1500 mg daily
Melatonin 2 - 3 mg daily, taken 2 hrs or less before bedtime
RNA and DNA see label
SOD (superoxide dismutase) see label
Copper 3 mg
Vitamin C with bioflavonoids 6000 - 10000 mg daily in divided doses.
Alzheimer's Disease
More Information Found In This Column
Dementia
May result from arteriosclerosis that slowly cuts off
the supply of blood the brain. The death of brain
tissue form a series of minor strokes, or from the
pressure exerted by an accumulation of fluid in the
brain, may cause dementia.
Blood clots (small)        
The presence of small blood clots in vessels that
supply the brain, a brain tumor, hypothyroidism, and
advanced syphilis all can cause symptoms similar to
those of Alzheimer’s disease
Drugs OTC & Prescribed        
The average person over the age of sixty five is likely
to be taking between eight and ten different
prescription and over-the-counter drugs. Drug
reactions, coupled with a nutrient-poor diet, often
adversely affect people not only physically, but
mentally.
Nutritional deficiencies       
The precise cause of Alzheimer’s disease is unknown,
but research reveals a number of interesting clues.
Many of them point to nutritional deficiencies. For
example people with Alzheimer’s tend to have low
levels of vitamin B12, B3, and zinc in their bodies.
The B vitamins are important in cognitive
functioning. The development of neurofibrillary
tangles and amyloid plaques in the brain that are
characteristic of the disease have been associated
with zinc deficiency. Malabsorption problems, which
are common among elderly people, make them more
prone than others to nutritional deficiencies. Alcohol
and many medications further deplete crucial
vitamins and minerals. Low levels of antioxidant
vitamins A and E and the Carotenoids (including beta-
carotene) are found in people with Alzheimer’s
disease. These nutrients act as free radical
scavengers, deficiencies may expose the brain cells to
increased oxidative damage. Deficiencies of baron,
potassium, and selenium have been found in people
with Alzheimer’s disease.
Aluminum         
Autopsies of people who have died of Alzheimer's
disease reveal excessive amounts of aluminum in the
hippocampus area of the cerebral cortex, the external
layer of the gray matter responsible for higher brain
functions such as abstract thinking, judgment,
memory, and language. Aluminum can be found in
many food sources including white flour.
Mercury        
Brains of people with Alzheimer’s disease have been
found to contain higher than normal concentrations
of the toxic metal mercury. For most people, the
release of mercury from dental amalgams is the
primary means of mercury exposure.  Mercury is also
found as a preservative in vaccinations.
Heredity        
At least four gene variations are linked to Alzheimer’s
disease. All of them reduce the clearance, or increase
the production, of beta-amyloid.
Immune system        
Many illnesses result from immune system malfunction
causing it to attack the body’s own tissues. Powerful
immune system proteins called complement proteins
have been found around the plaques and tangles in
the brains of people who have died of Alzheimer’s
disease.
Brain injury       
In animals, brain injury is known to result in a
alteration in the genetic “instructions” for two kinds
of complement proteins. Complement proteins
normally help clear away dead cells but at some point
they may begin to attack healthy cells as well. Further
evidence reveals that the presence of amyloid may
trigger the release of a cascade of complement
proteins, perhaps sparking a viscous cycle of
inflammation and further plaque deposits.
Other disorders with symptoms similar
to those of Alzheimer’s disease:
Dr. Vincent Fortanasce has assembled a
very intesive and detailed diet plan for
Alzheimer's Disease. He has put a lot of
accurate work into it,
so check it out.